The pathophysiological mechanism underlying Brugada syndrome. Depolarization versus repolarization

Arthur A.M. Wilde; Pieter G. Postema; José M. Di Diego; Sami Viskin; Hiroshi Morita; Jeffrey M. Fish; Charles Antzelevitch

doi:10.1016/j.yjmcc.2010.07.012

The pathophysiological mechanism underlying Brugada syndrome. Depolarization versus repolarization

Arthur A.M. Wilde, Pieter G. Postema, José M. Di Diego, Sami Viskin, Hiroshi Morita, Jeffrey M. Fish, Charles Antzelevitch^*

^*Corresponding author for this work

Research output: Contribution to journal › Comment/debate

305 Scopus citations

Abstract

This Point/Counterpoint presents a scholarly debate of the mechanisms underlying the electrocardiographic and arrhythmic manifestations of Brugada syndrome (BrS), exploring in detail the available evidence in support of the repolarization vs. depolarization hypothesis.

Original language	English
Pages (from-to)	543-553
Number of pages	11
Journal	Journal of Molecular and Cellular Cardiology
Volume	49
Issue number	4
DOIs	https://doi.org/10.1016/j.yjmcc.2010.07.012
State	Published - Oct 2010
Externally published	Yes

Keywords

Brugada syndrome
Depolarization versus repolarization

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10.1016/j.yjmcc.2010.07.012

Cite this

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abstract = "This Point/Counterpoint presents a scholarly debate of the mechanisms underlying the electrocardiographic and arrhythmic manifestations of Brugada syndrome (BrS), exploring in detail the available evidence in support of the repolarization vs. depolarization hypothesis.",

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The pathophysiological mechanism underlying Brugada syndrome. Depolarization versus repolarization

Abstract

Keywords

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