The neurological phenotype of ataxia-telangiectasia: Solving a persistent puzzle

Keyphrases

• Ataxia Telangiectasia 100%
• DNA Damage Response 100%
• Neurological Phenotype 100%
• Genomic Instability 40%
• Protein Kinase 20%
• Vulnerability 20%
• Major Features 20%
• Normal Life 20%
• Life Conditions 20%
• Mouse Model 20%
• Nervous System 20%
• Human Genomics 20%
• Cellular Response 20%
• Neurodegeneration 20%
• Immunodeficiency 20%
• Degree of Severity 20%
• Ionizing Radiation 20%
• ATM Protein 20%
• Proliferating Cells 20%
• Genome Integrity 20%
• Double-strand Breaks 20%
• Radiomimetic 20%
• Neuronal Phenotype 20%
• Neuron Cells 20%
• Cancer Predisposition 20%
• CNS Development 20%
• Mode of Death 20%
• Postmitotic Neurons 20%
• Multi-branch 20%

Neuroscience

• DNA Damage Response 100%
• Ataxia Telangiectasia 100%
• Nervous System 20%
• Cell Proliferation 20%
• ATM Protein 20%
• Nerve Cell Degeneration 20%

Biochemistry, Genetics and Molecular Biology

• DNA Damage Response 100%
• Genome Instability 40%
• Mouse Model 20%
• Protein Kinases 20%
• Human Genomics 20%
• Cell Proliferation 20%
• Nerve Cell Degeneration 20%
• Genomics 20%