Neuroprotective therapy for argon-laser induced retinal injury

M. Belkin*, M. Rosner, Y. Solberg, Y. Turetz

*Corresponding author for this work

Research output: Contribution to journalConference articlepeer-review

Abstract

The efficacy of neuroprotective compounds in ameliorating laser-induced retinal damage was studied in a rat model. The laser injury mainly involved the outer layers of the retina, where it destroyed significant numbers of photoreceptor cells. Over time, evidence of two major histopathological processes was observed: traction of adjacent normal retinal cells into the central area of the lesion forming an internal retinal bulging; and a retinal pigmented epithelial proliferative reaction associated with subretinal neovascularization and invasion of the retinal lesion site by phagocytes. The neuroprotective compounds studied were corticosteroid methylprednisolone, glutamate-receptor blocker MK-801, antioxidant enzyme superoxide dismutase, and flunarizine.

Original languageEnglish
Pages (from-to)407-421
Number of pages15
JournalProceedings of SPIE - The International Society for Optical Engineering
Volume3591
DOIs
StatePublished - 1999
EventProceedings of the 1999 Ophthalmic Technologies IX - San Jose, CA, USA
Duration: 23 Jan 199925 Jan 1999

Funding

FundersFunder number
Bloom's Syndrome Foundation91-00225

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