Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Nili Naftali-Shani; Natali Molotski; Yael Nevo-Caspi; Michael Arad; Rafael Kuperstein; Uri Amit; Irit Huber; Li at Zeltzer; Alina Levich; Haya Abbas; Lorenzo Monserrat; Gideon Paret; Jonathan Leor

doi:10.1161/CIRCULATIONAHA.118.035950

Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Nili Naftali-Shani, Natali Molotski, Yael Nevo-Caspi, Michael Arad, Rafael Kuperstein, Uri Amit, Irit Huber, Li at Zeltzer, Alina Levich, Haya Abbas, Lorenzo Monserrat, Gideon Paret, Jonathan Leor^*

^*Corresponding author for this work

Research output: Contribution to journal › Letter › peer-review

8 Scopus citations

Original language	English
Pages (from-to)	2721-2723
Number of pages	3
Journal	Circulation
Volume	138
Issue number	23
DOIs	https://doi.org/10.1161/CIRCULATIONAHA.118.035950
State	Published - 2018

Funding

Funders	Funder number
Imperial Tobacco Ltd.

Keywords

Cardiomyopathies
Induced pluripotent stem cell
Inflammation
Neovascularization physiologic
STAT3
Vascular endothelial growth factors

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

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10.1161/CIRCULATIONAHA.118.035950

Cite this

Naftali-Shani, N., Molotski, N., Nevo-Caspi, Y., Arad, M., Kuperstein, R., Amit, U., Huber, I., Zeltzer, L. A., Levich, A., Abbas, H., Monserrat, L., Paret, G., & Leor, J. (2018). Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes. Circulation, 138(23), 2721-2723. https://doi.org/10.1161/CIRCULATIONAHA.118.035950

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title = "Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes",

keywords = "Cardiomyopathies, Induced pluripotent stem cell, Inflammation, Neovascularization physiologic, STAT3, Vascular endothelial growth factors",

author = "Nili Naftali-Shani and Natali Molotski and Yael Nevo-Caspi and Michael Arad and Rafael Kuperstein and Uri Amit and Irit Huber and Zeltzer, {Li at} and Alina Levich and Haya Abbas and Lorenzo Monserrat and Gideon Paret and Jonathan Leor",

note = "Funding Information: This project was supported by a research grant from Imperial Tobacco Ltd.",

year = "2018",

doi = "10.1161/CIRCULATIONAHA.118.035950",

language = "אנגלית",

volume = "138",

pages = "2721--2723",

journal = "Circulation",

issn = "0009-7322",

publisher = "Lippincott Williams and Wilkins",

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}

Naftali-Shani, N, Molotski, N, Nevo-Caspi, Y, Arad, M , Kuperstein, R, Amit, U, Huber, I, Zeltzer, LA, Levich, A, Abbas, H, Monserrat, L, Paret, G & Leor, J 2018, 'Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes', Circulation, vol. 138, no. 23, pp. 2721-2723. https://doi.org/10.1161/CIRCULATIONAHA.118.035950

TY - JOUR

T1 - Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

AU - Naftali-Shani, Nili

AU - Molotski, Natali

AU - Nevo-Caspi, Yael

AU - Arad, Michael

AU - Kuperstein, Rafael

AU - Amit, Uri

AU - Huber, Irit

AU - Zeltzer, Li at

AU - Levich, Alina

AU - Abbas, Haya

AU - Monserrat, Lorenzo

AU - Paret, Gideon

AU - Leor, Jonathan

N1 - Funding Information: This project was supported by a research grant from Imperial Tobacco Ltd.

PY - 2018

Y1 - 2018

KW - Cardiomyopathies

KW - Induced pluripotent stem cell

KW - Inflammation

KW - Neovascularization physiologic

KW - STAT3

KW - Vascular endothelial growth factors

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U2 - 10.1161/CIRCULATIONAHA.118.035950

DO - 10.1161/CIRCULATIONAHA.118.035950

M3 - ???researchoutput.researchoutputtypes.contributiontojournal.letter???

AN - SCOPUS:85058915965

SN - 0009-7322

VL - 138

SP - 2721

EP - 2723

JO - Circulation

JF - Circulation

IS - 23

ER -

Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Funding

Keywords

UN SDGs

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