Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Nili Naftali-Shani; Natali Molotski; Yael Nevo-Caspi; Michael Arad; Rafael Kuperstein; Uri Amit; Irit Huber; Li at Zeltzer; Alina Levich; Haya Abbas; Lorenzo Monserrat; Gideon Paret; Jonathan Leor

doi:10.1161/CIRCULATIONAHA.118.035950

Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Nili Naftali-Shani, Natali Molotski, Yael Nevo-Caspi, Michael Arad, Rafael Kuperstein, Uri Amit, Irit Huber, Li at Zeltzer, Alina Levich, Haya Abbas, Lorenzo Monserrat, Gideon Paret, Jonathan Leor^*

^*Corresponding author for this work

Research output: Contribution to journal › Letter › peer-review

8 Scopus citations

Original language	English
Pages (from-to)	2721-2723
Number of pages	3
Journal	Circulation
Volume	138
Issue number	23
DOIs	https://doi.org/10.1161/CIRCULATIONAHA.118.035950
State	Published - 2018

Keywords

Cardiomyopathies
Induced pluripotent stem cell
Inflammation
Neovascularization physiologic
STAT3
Vascular endothelial growth factors

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

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10.1161/CIRCULATIONAHA.118.035950

Cite this

Naftali-Shani, N., Molotski, N., Nevo-Caspi, Y., Arad, M., Kuperstein, R., Amit, U., Huber, I., Zeltzer, L. A., Levich, A., Abbas, H., Monserrat, L., Paret, G., & Leor, J. (2018). Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes. Circulation, 138(23), 2721-2723. https://doi.org/10.1161/CIRCULATIONAHA.118.035950

@article{b2f7be8796fc4abbb3f29252d31f5f67,

title = "Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes",

keywords = "Cardiomyopathies, Induced pluripotent stem cell, Inflammation, Neovascularization physiologic, STAT3, Vascular endothelial growth factors",

author = "Nili Naftali-Shani and Natali Molotski and Yael Nevo-Caspi and Michael Arad and Rafael Kuperstein and Uri Amit and Irit Huber and Zeltzer, {Li at} and Alina Levich and Haya Abbas and Lorenzo Monserrat and Gideon Paret and Jonathan Leor",

note = "Funding Information: This project was supported by a research grant from Imperial Tobacco Ltd.",

year = "2018",

doi = "10.1161/CIRCULATIONAHA.118.035950",

language = "אנגלית",

volume = "138",

pages = "2721--2723",

journal = "Circulation",

issn = "0009-7322",

publisher = "Lippincott Williams and Wilkins Ltd.",

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}

Naftali-Shani, N, Molotski, N, Nevo-Caspi, Y, Arad, M , Kuperstein, R, Amit, U, Huber, I, Zeltzer, LA, Levich, A, Abbas, H, Monserrat, L, Paret, G & Leor, J 2018, 'Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes', Circulation, vol. 138, no. 23, pp. 2721-2723. https://doi.org/10.1161/CIRCULATIONAHA.118.035950

TY - JOUR

T1 - Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

AU - Naftali-Shani, Nili

AU - Molotski, Natali

AU - Nevo-Caspi, Yael

AU - Arad, Michael

AU - Kuperstein, Rafael

AU - Amit, Uri

AU - Huber, Irit

AU - Zeltzer, Li at

AU - Levich, Alina

AU - Abbas, Haya

AU - Monserrat, Lorenzo

AU - Paret, Gideon

AU - Leor, Jonathan

N1 - Funding Information: This project was supported by a research grant from Imperial Tobacco Ltd.

PY - 2018

Y1 - 2018

KW - Cardiomyopathies

KW - Induced pluripotent stem cell

KW - Inflammation

KW - Neovascularization physiologic

KW - STAT3

KW - Vascular endothelial growth factors

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U2 - 10.1161/CIRCULATIONAHA.118.035950

DO - 10.1161/CIRCULATIONAHA.118.035950

M3 - ???researchoutput.researchoutputtypes.contributiontojournal.letter???

AN - SCOPUS:85058915965

SN - 0009-7322

VL - 138

SP - 2721

EP - 2723

JO - Circulation

JF - Circulation

IS - 23

ER -

Modeling peripartum cardiomyopathy with human induced pluripotent stem cells reveals distinctive abnormal function of cardiomyocytes

Keywords

UN SDGs

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