Late prenatal immune activation in mice leads to behavioral and neurochemical abnormalities relevant to the negative symptoms of schizophrenia

Keyphrases

• Neurochemicals 100%
• Prenatal Immune Activation 100%
• Negative Symptoms of Schizophrenia 100%
• Prefrontal Cortex 66%
• Schizophrenia 66%
• Hippocampus 66%
• Poly IC 66%
• Female Offspring 66%
• Prenatal Immune Challenge 66%
• Glycine 33%
• Behavioral Response 33%
• Dopamine 33%
• Latent Inhibition 33%
• Neurodevelopmental 33%
• Male Offspring 33%
• Perinatal Infection 33%
• Insults 33%
• Gestational Day 33%
• Polyinosinic-polycytidylic Acid 33%
• Sex-specific 33%
• Viral 33%
• Prenatal Exposure 33%
• Cognitive Impairment 33%
• Inhibition Effect 33%
• Apomorphine 33%
• Locomotor 33%
• Glutamate 33%
• Vulnerability 33%
• High Risk 33%
• Social Interaction 33%
• Behavioral Consequences 33%
• Infection Risk 33%
• Aminobutyric Acid 33%
• Precise Timing 33%
• C(V) 33%
• Late Gestation 33%
• Epidemiological Association 33%
• Neurotransmitter Levels 33%
• Anhedonic Behavior 33%

Neuroscience

• Polyinosinic:polycytidylic Acid 100%
• Negative Syndrome 100%
• Prefrontal Cortex 66%
• Hippocampus 66%
• Gamma-Aminobutyric Acid 66%
• Behavior (Neuroscience) 33%
• Latent Inhibition 33%
• Apomorphine 33%
• Neurotransmitter 33%
• Glutamic Acid 33%
• Social Interaction 33%