Abstract
It is proposed that interferon is a specifically oxidized, disulfide form of initiation factor(s); that it arises as a byproduct of inhibition of the initiation of protein synthesis by certain compounds; that is derives its specificity in extracellular form from specific glycosylation and; that it is directly responsible for antiviral activity. The hypothesis is based on a new interpretation of the extensive literature on interferon.
| Original language | English |
|---|---|
| Pages (from-to) | 11-17 |
| Number of pages | 7 |
| Journal | J.MOLEC.MED. |
| Volume | 1 |
| Issue number | 1 |
| State | Published - 1975 |