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Hormonal interactions in human prostate tumor LNCaP cells
Zippora Lupowitz,
Nava Zisapel
*
*
Corresponding author for this work
Department of Neurobiology
Tel Aviv University
Research output
:
Contribution to journal
›
Article
›
peer-review
36
Scopus citations
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Keyphrases
Melatonin
100%
Prostate Tumor
100%
LNCaP Cells
100%
Human Prostate
100%
Hormonal Stimulation
100%
Estradiol
62%
Cell Growth
25%
3H-thymidine Incorporation
25%
Androgen-independent
25%
Prostate Epithelial Cells
25%
Attenuation
12%
Estrogen
12%
Cell Viability
12%
Protein Kinase
12%
DNA Content
12%
Cell Support
12%
Pineal Gland
12%
Physiological Concentrations
12%
Direct Interaction
12%
Protein Kinase C Inhibitor
12%
LNCaP
12%
Tumor Cell Lines
12%
Melatonin Receptors
12%
Prostatic Tumor
12%
Benign Prostate Disease
12%
Prostate Tumor Cells
12%
Neuroscience
Melatonin
100%
Prostate
100%
Diethylstilbestrol
62%
Cell Growth
25%
DNA Synthesis
25%
Protein Kinase C
25%
Androgen
25%
Epithelial Cells
25%
Epileptic Absence
12%
Cell Line
12%
Cell Viability
12%
Melatonin Receptor
12%
Biochemistry, Genetics and Molecular Biology
Melatonin
100%
LNCaP
100%
Estradiol
62%
DNA Synthesis
25%
Protein Kinase C
25%
Cell Growth
25%
DNA Content
12%
Cell Viability
12%
Melatonin Receptor
12%
Pharmacology, Toxicology and Pharmaceutical Science
Melatonin
100%
Prostate Tumor
100%
Diethylstilbestrol
62%
Androgen
25%
Thymidine
25%
Epileptic Absence
12%
Protein Kinase C
12%
Cell Viability
12%
Protein Kinase C Inhibitor
12%
Melatonin Receptor
12%