GM1 ganglioside prevents axonal regeneration inhibition and cognitive deficits in a mouse model of traumatic brain injury

Keyphrases

• Cognitive Deficits 100%
• Inhibition Deficit 100%
• Mild Traumatic Brain Injury (mTBI) 100%
• Mouse Model 100%
• Traumatic Brain Injury 100%
• Axonal Regeneration 100%
• GM1 Ganglioside 100%
• Tubulin 50%
• Trauma 50%
• Novel Object Recognition 50%
• Spatial Memory 50%
• Cortex 50%
• Brain Injury 50%
• Y-maze 50%
• Drop Weight 50%
• Cognitive Change 50%
• Immunostaining 50%
• Most Common Cause 50%
• Biochemical Changes 50%
• Potential Interventions 50%
• Axon 50%
• Cellular Changes 50%
• Neuronal Survival 50%
• Neuroprotective 50%
• Filamentous Actin (F-actin) 50%
• Induced Damage 50%
• Young Population 50%
• Gangliosides 50%
• NeuN 50%
• Outgrowth Inhibition 50%
• Axonal Outgrowth 50%
• Traumatic Brain Injury Models 50%
• Growth Cone 50%
• PNfH 50%
• Closed Head 50%
• Visual Memory 50%
• Neurological Damage 50%

Neuroscience

• Cognitive Disorders 100%
• Neurotrauma 100%
• Traumatic Brain Injury 100%
• Ganglioside GM1 100%
• Neuroprotective Agent 33%
• Brain Injury 33%
• Visual Memory 33%
• Cognitive Change 33%
• Immunohistochemistry 33%
• Actin 33%
• Growth Cone 33%
• Tubulin 33%
• NeuN 33%
• Ganglioside 33%
• Axon 33%