Glycinuria, a hereditary disorder associated with nephrolithiasis

André de Vries; Shaul Kochwa; Jacob Lazebnik; Menahem Frank; Meir Djaldetti

doi:10.1016/0002-9343(57)90320-0

Glycinuria, a hereditary disorder associated with nephrolithiasis

André de Vries^*, Shaul Kochwa, Jacob Lazebnik, Menahem Frank, Meir Djaldetti

^*Corresponding author for this work

Clinical Departments

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Abstract

Excessive urinary glycine excretion was found in four members of a family and was associated in three members with nephrolithiasis. The glycinuria was due to a renal mechanism. Failure to reabsorb glycine was not associated with defective reabsorption of other amino acids or of phosphate or glucose. A kidney stone obtained from one of these patients was composed mainly of calcium oxalate and contained a small amount of glycine present in non-protein, non-peptide form.

Original language	English
Pages (from-to)	408-415
Number of pages	8
Journal	American Journal of Medicine
Volume	23
Issue number	3
DOIs	https://doi.org/10.1016/0002-9343(57)90320-0
State	Published - Sep 1957

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title = "Glycinuria, a hereditary disorder associated with nephrolithiasis",

abstract = "Excessive urinary glycine excretion was found in four members of a family and was associated in three members with nephrolithiasis. The glycinuria was due to a renal mechanism. Failure to reabsorb glycine was not associated with defective reabsorption of other amino acids or of phosphate or glucose. A kidney stone obtained from one of these patients was composed mainly of calcium oxalate and contained a small amount of glycine present in non-protein, non-peptide form.",

author = "\{de Vries\}, Andr{\'e} and Shaul Kochwa and Jacob Lazebnik and Menahem Frank and Meir Djaldetti",

year = "1957",

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T1 - Glycinuria, a hereditary disorder associated with nephrolithiasis

AU - de Vries, André

AU - Kochwa, Shaul

AU - Lazebnik, Jacob

AU - Frank, Menahem

AU - Djaldetti, Meir

PY - 1957/9

Y1 - 1957/9

N2 - Excessive urinary glycine excretion was found in four members of a family and was associated in three members with nephrolithiasis. The glycinuria was due to a renal mechanism. Failure to reabsorb glycine was not associated with defective reabsorption of other amino acids or of phosphate or glucose. A kidney stone obtained from one of these patients was composed mainly of calcium oxalate and contained a small amount of glycine present in non-protein, non-peptide form.

AB - Excessive urinary glycine excretion was found in four members of a family and was associated in three members with nephrolithiasis. The glycinuria was due to a renal mechanism. Failure to reabsorb glycine was not associated with defective reabsorption of other amino acids or of phosphate or glucose. A kidney stone obtained from one of these patients was composed mainly of calcium oxalate and contained a small amount of glycine present in non-protein, non-peptide form.

UR - https://www.scopus.com/pages/publications/49749154084

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JO - American Journal of Medicine

JF - American Journal of Medicine

IS - 3

ER -

Glycinuria, a hereditary disorder associated with nephrolithiasis

Abstract

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