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Gene silencing at the nuclear periphery
Sigal Shaklai
, Ninette Amariglio
,
Gideon Rechavi
, Amos J. Simon
*
*
Corresponding author for this work
Internal Medicine
Hematology
Sheba Medical Center at Tel Hashomer
Tel Aviv University
Research output
:
Contribution to journal
›
Short survey
›
peer-review
90
Scopus citations
Overview
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Keyphrases
Aberrant Gene Expression
25%
Bone Morphogenetic Protein
25%
Chromatin Modifiers
50%
Cumulative Data
25%
Direct Binding
25%
Emerin
50%
Epigenetics
25%
Gene Regulation
25%
Gene Repression
25%
Gene Silencing
100%
Genetic Disease
25%
Heterochromatin
25%
Histone Acetylation
25%
Histone Code
25%
Histone Post-translational Modifications
25%
Inner nuclear Membrane
25%
Lamin
50%
Lamin B Receptor
25%
Lamina Association
25%
Laminopathy
25%
Major Challenges
25%
MAN1
25%
Modifier Factors
25%
Molecular Mechanism
25%
MyoD
25%
Nuclear Envelopathies
25%
Nuclear Envelope
100%
Nuclear Envelope Proteins
25%
Nuclear Lamina
50%
Nuclear Periphery
100%
Nuclear Pore Complex
25%
Outer nuclear Membrane
25%
Polypeptide
25%
Retinoblastoma 1
25%
Signaling Pathway
25%
Support Structure
25%
Transcription Factor
50%
Transcriptional Regulation
25%
Transcriptional Repression
25%
Transcriptional Repressor
25%
Transforming Growth Factor-β
25%
Biochemistry, Genetics and Molecular Biology
Bone Morphogenetic Protein
20%
Chromatin
40%
Deacetylation
20%
Downregulation
20%
E2F
20%
Emerin
40%
Gene Expression
40%
Gene Inactivation
100%
Gene Repression
20%
Genetic Disorder
20%
Heterochromatin
20%
Histone
20%
Histone Code
20%
Histone Modification
20%
Lamin B Receptor
20%
Molecular Mechanism
20%
MyoD
20%
Nuclear Lamina
40%
Nuclear Membrane
100%
Nuclear Pore
100%
Nuclear Pore Complex
20%
Retinoblastoma
20%
Signal Transduction
20%
Transcription
40%
Transcription Factors
40%
Transcriptional Repressor
20%
Transforming Growth Factor Beta
20%