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Defects in insulin's signal transduction in old rat livers
Orna Nadiv
,
Ohad Cohen
, Yehiel Zick
*
*
Corresponding author for this work
Weizmann Institute of Science
Rabin Medical Center Israel
Research output
:
Contribution to journal
›
Article
›
peer-review
25
Scopus citations
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Keyphrases
Aged Rats
100%
Insulin Receptor Kinase
100%
Rat Liver
100%
Insulin Signal Transduction
100%
Kinase Activity
75%
Phosphorylation
37%
Insulin
25%
Aging
25%
Glucose Intolerance
25%
Young Rats
25%
In Vivo Activity
25%
Old Animals
25%
Immunoblotting
12%
Wistar Rats
12%
Severe Disease
12%
Diabetes
12%
Age-related
12%
Tyrosine Phosphorylation
12%
Insulin Receptor
12%
Autophosphorylation
12%
Insulin Action
12%
Maximal Effect
12%
Anti-P
12%
Acquired Defects
12%
Physiological Substrates
12%
Dose-dependent
12%
Neuroscience
Insulin Receptor Kinase
100%
Signal Transduction
100%
In Vitro
37%
Insulin
37%
phosphorylation
37%
In Vivo
25%
Glucose
25%
Protein Phosphorylation
12%
Insulin Receptor
12%
Receptor
12%
Immunoblotting
12%
Pharmacology, Toxicology and Pharmaceutical Science
Insulin Receptor Kinase
100%
Rat
100%
Insulin
37%
Glucose Intolerance
25%
Western Blot
12%
Receptor
12%
Wistar Rat
12%
Tyrosine
12%
Insulin Receptor
12%