Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4

Maayan Waldman; Keren Cohen; Dor Yadin; Vadim Nudelman; Dan Gorfil; Michal Laniado-Schwartzman; Ran Kornwoski; Dan Aravot; Nader G. Abraham; Michael Arad; Edith Hochhauser

doi:10.1186/s12933-018-0757-1

Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4

Maayan Waldman
, Keren Cohen
, Dor Yadin
, Vadim Nudelman
, Dan Gorfil
, Michal Laniado-Schwartzman
, Ran Kornwoski
, Dan Aravot
, Nader G. Abraham
, Michael Arad
, Edith Hochhauser^*

^*Corresponding author for this work

Research output: Contribution to journal › Comment/debate

8 Scopus citations

Abstract

Unfortunately, after publication of this article [1], it was noticed that Table1 contained errors introduced during the production process. In the WT+AT column, the FS value is 21±7 and the Body Weight value is 25±2. In the WT+AT+CR column, the FS value is 46±14 and the Body Weight value is 19±1. The original article has been updated to reflect this.

Original language	English
Article number	115
Journal	Cardiovascular Diabetology
Volume	17
Issue number	1
DOIs	https://doi.org/10.1186/s12933-018-0757-1
State	Published - 17 Aug 2018

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Waldman, M., Cohen, K., Yadin, D., Nudelman, V., Gorfil, D., Laniado-Schwartzman, M., Kornwoski, R., Aravot, D., Abraham, N. G., Arad, M., & Hochhauser, E. (2018). Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4. Cardiovascular Diabetology, 17(1), Article 115. https://doi.org/10.1186/s12933-018-0757-1

@article{b993237270ec48ee99dfc1bb6b168c06,

title = "Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4",

abstract = "Unfortunately, after publication of this article [1], it was noticed that Table1 contained errors introduced during the production process. In the WT+AT column, the FS value is 21±7 and the Body Weight value is 25±2. In the WT+AT+CR column, the FS value is 46±14 and the Body Weight value is 19±1. The original article has been updated to reflect this.",

author = "Maayan Waldman and Keren Cohen and Dor Yadin and Vadim Nudelman and Dan Gorfil and Michal Laniado-Schwartzman and Ran Kornwoski and Dan Aravot and Abraham, \{Nader G.\} and Michael Arad and Edith Hochhauser",

note = "Publisher Copyright: {\textcopyright} 2018 The Author(s).",

year = "2018",

month = aug,

day = "17",

doi = "10.1186/s12933-018-0757-1",

language = "אנגלית",

volume = "17",

journal = "Cardiovascular Diabetology",

issn = "1475-2840",

publisher = "BioMed Central Ltd.",

number = "1",

}

Waldman, M, Cohen, K, Yadin, D, Nudelman, V, Gorfil, D, Laniado-Schwartzman, M, Kornwoski, R, Aravot, D, Abraham, NG, Arad, M & Hochhauser, E 2018, 'Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4', Cardiovascular Diabetology, vol. 17, no. 1, 115. https://doi.org/10.1186/s12933-018-0757-1

Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4. / Waldman, Maayan; Cohen, Keren; Yadin, Dor et al.
In: Cardiovascular Diabetology, Vol. 17, No. 1, 115, 17.08.2018.

Research output: Contribution to journal › Comment/debate

TY - JOUR

T1 - Correction

T2 - Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4

AU - Waldman, Maayan

AU - Cohen, Keren

AU - Yadin, Dor

AU - Nudelman, Vadim

AU - Gorfil, Dan

AU - Laniado-Schwartzman, Michal

AU - Kornwoski, Ran

AU - Aravot, Dan

AU - Abraham, Nader G.

AU - Arad, Michael

AU - Hochhauser, Edith

PY - 2018/8/17

Y1 - 2018/8/17

N2 - Unfortunately, after publication of this article [1], it was noticed that Table1 contained errors introduced during the production process. In the WT+AT column, the FS value is 21±7 and the Body Weight value is 25±2. In the WT+AT+CR column, the FS value is 46±14 and the Body Weight value is 19±1. The original article has been updated to reflect this.

AB - Unfortunately, after publication of this article [1], it was noticed that Table1 contained errors introduced during the production process. In the WT+AT column, the FS value is 21±7 and the Body Weight value is 25±2. In the WT+AT+CR column, the FS value is 46±14 and the Body Weight value is 19±1. The original article has been updated to reflect this.

UR - https://www.scopus.com/pages/publications/85051990517

U2 - 10.1186/s12933-018-0757-1

DO - 10.1186/s12933-018-0757-1

M3 - ???researchoutput.researchoutputtypes.contributiontojournal.comment???

AN - SCOPUS:85051990517

SN - 1475-2840

VL - 17

JO - Cardiovascular Diabetology

JF - Cardiovascular Diabetology

IS - 1

M1 - 115

ER -

Waldman M, Cohen K, Yadin D, Nudelman V, Gorfil D, Laniado-Schwartzman M et al. Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4. Cardiovascular Diabetology. 2018 Aug 17;17(1):115. doi: 10.1186/s12933-018-0757-1

Correction: Regulation of diabetic cardiomyopathy by caloric restriction is mediated by intracellular signaling pathways involving 'SIRT1 and PGC-1α'. [Cardiovasc Diabetol., 17, (2018) (111)] DOI: 10.1186/s12933-018-0754-4

Abstract

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