Childhood ataxia with CNS hypomyelination/vanishing white matter disease-A common leukodystrophy caused by abnormal control of protein synthesis

Keyphrases

• Increased Vulnerability 100%
• Protein Synthesis 100%
• Hypomyelination 100%
• Vanishing White Matter Disease 100%
• Leukodystrophy 100%
• Childhood Ataxia 100%
• Eukaryotic Initiation Factor-2B 100%
• Astrocytes 50%
• Neuropathology 50%
• Dementia 50%
• Physiological Stress 50%
• Stress Conditions 50%
• EIF2 50%
• Endoplasmic Reticulum Stress (ER stress) 50%
• Activating Transcription Factor 4 (ATF4) 50%
• Protein Translation 50%
• Glial Cells 50%
• Oligodendrocyte 50%
• Cerebrospinal Fluid 50%
• White Matter Abnormalities 50%
• Understanding Why 50%
• Biochemical Abnormalities 50%
• Eukaryotic Translation Initiation Factor 2B 50%
• Adult-onset Ataxia 50%
• Ovarian Insufficiency 50%
• Juvenile-onset 50%
• Asialotransferrin 50%
• Prenatal Origin 50%
• White Matter Disease 50%
• Neurological Abnormalities 50%
• Guanine nucleotide Exchange 50%

Neuroscience

• Protein Biosynthesis 100%
• White Matter Disease 100%
• Ataxia 100%
• Guanine Nucleotide Exchange Factor 100%
• Leukodystrophy 100%
• Astrocyte 33%
• Magnetic Resonance Imaging 33%
• Endoplasmic Reticulum Stress 33%
• Oligodendrocyte 33%
• Translation Initiation 33%
• Guanine Nucleotide 33%
• Transferrin 33%
• Glial Cells 33%
• ATF4 33%

Biochemistry, Genetics and Molecular Biology

• Protein Biosynthesis 100%
• Guanine Nucleotide Exchange Factor 100%
• Astrocyte 33%
• Endoplasmic Reticulum Stress 33%
• ATF4 33%
• Translation (Protein Synthesis) 33%
• Guanine 33%
• Physiological Stress 33%
• Liquid 33%
• Eukaryotic Translation 33%
• Initiation Factor 33%
• Glial Cells 33%
• Transferrin 33%
• Oligodendrocyte 33%