TY - JOUR
T1 - Catheter Atrioventricular Junctional Ablation in Patients with Accessory Pathways
AU - ELDAR, MICHAEL
AU - GRIFFIN, JERRY C.
AU - SEGER, JOHN J.
AU - ABBOTT, JOSEPH A.
AU - RUDER, MICHAEL A.
AU - DAVIS, JESSE C.
AU - HERRE, JOHN M.
AU - SCHEINMAN, MELVIN M.
PY - 1986/11
Y1 - 1986/11
N2 - Seven patients with accessory pathway and symptomatic alrioventricular reciprocating tachycardia underwent catheter ablution of the atrioventricular junction (AVJ). Four patients had the Wolff‐Park inson White syndrome, two had concealed left free‐wall accessory pathways, and one patient had a nodoventriculor connection. All patients failed multiple antiarrhythmic drugs and one failed attempted surgical ablation of a posteroseptal accessory pathway. Chronic interruption of atrioventricular node‐His conduction was achieved in all patients. Over a mean follow‐up period of 21 ± 14 months, four patients remained asymptomatic without antiarrhythmic therapy. One patient developed atrial fibrillation after magnet application to her VVI pacemaker, another developed atrial gutter, and a third had nonparoxysmal sinus or atrial tachycardia. Two patients required chronic quinidine therapy. Two patients with concealed accessory pathways had pacemaker‐mediated tachycardia which was controlled by pacemaker reprogramming. Atrioventricular junctional ablation in patients with accessory pathways proved elective in that all are currently controlled without need for surgical intervention. On follow‐up, a relatively high incidence of atrial arrhythmias requiring antiarrhythmic therapy was found.
AB - Seven patients with accessory pathway and symptomatic alrioventricular reciprocating tachycardia underwent catheter ablution of the atrioventricular junction (AVJ). Four patients had the Wolff‐Park inson White syndrome, two had concealed left free‐wall accessory pathways, and one patient had a nodoventriculor connection. All patients failed multiple antiarrhythmic drugs and one failed attempted surgical ablation of a posteroseptal accessory pathway. Chronic interruption of atrioventricular node‐His conduction was achieved in all patients. Over a mean follow‐up period of 21 ± 14 months, four patients remained asymptomatic without antiarrhythmic therapy. One patient developed atrial fibrillation after magnet application to her VVI pacemaker, another developed atrial gutter, and a third had nonparoxysmal sinus or atrial tachycardia. Two patients required chronic quinidine therapy. Two patients with concealed accessory pathways had pacemaker‐mediated tachycardia which was controlled by pacemaker reprogramming. Atrioventricular junctional ablation in patients with accessory pathways proved elective in that all are currently controlled without need for surgical intervention. On follow‐up, a relatively high incidence of atrial arrhythmias requiring antiarrhythmic therapy was found.
KW - Wolff‐Parkinson‐White syndrome
KW - nodoventricular connection
KW - permanent pacemaker
KW - supraventricuJar tachycardia
UR - http://www.scopus.com/inward/record.url?scp=0022816134&partnerID=8YFLogxK
U2 - 10.1111/j.1540-8159.1986.tb06630.x
DO - 10.1111/j.1540-8159.1986.tb06630.x
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AN - SCOPUS:0022816134
SN - 0147-8389
VL - 9
SP - 810
EP - 820
JO - PACE - Pacing and Clinical Electrophysiology
JF - PACE - Pacing and Clinical Electrophysiology
IS - 6
ER -