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Antiphospholipid Syndrome
Nancy Agmon-Levin
*
, Angela Tincani
,
Yehuda Shoenfeld
*
Corresponding author for this work
Internal Medicine
Clinical Departments
University of Brescia
Research output
:
Chapter in Book/Report/Conference proceeding
›
Chapter
›
peer-review
Overview
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Keyphrases
Antiphospholipid Syndrome
100%
Antiphospholipid Antibodies
57%
β2-glycoprotein I (β2GPI)
28%
Environmental Factors
14%
Organ-specific
14%
Endothelial Cells
14%
Thrombosis
14%
Clinical Spectrum
14%
Infectious Agents
14%
Autoimmune Disease
14%
Monocytes
14%
Pregnancy Loss
14%
Cell Surface Receptor
14%
Intracellular Signaling
14%
Targeted Therapeutics
14%
Thrombotic
14%
Immune Complex
14%
Etiopathogenesis
14%
Vitamin D
14%
Trophoblast
14%
Therapeutic Interventions
14%
Immune-mediated Mechanisms
14%
Immunology and Microbiology
Antiphospholipid Syndrome
100%
Antiphospholipid Antibody
57%
Infectious Agent
14%
Cell Surface
14%
Monocyte
14%
Autoimmune Disease
14%
Intracellular Signaling
14%
Immune Complex
14%
Trophoblast
14%
Endothelial Cells
14%