Analysis of the relationships between ATM and the Rad54 paralogs involved in homologous recombination repair

Immunology and Microbiology

• Paralogy 100%
• Homologous Recombination 100%
• Recombination Repair 100%
• Hypersensitivity 40%
• Cross Linking 20%
• Mouse 20%
• Lifespan 20%
• Body Weight 20%
• Ataxia Telangiectasia 20%
• Immunodeficiency 20%
• Genomic Instability 20%
• Radioresistance 20%
• Mouse Strain 20%
• DNA Damage Response 20%
• DNA End Joining Repair 20%

Keyphrases

• Homologous Recombination Repair 100%
• Rad54 100%
• Homologous Recombination 22%
• Genotype 11%
• Functional Relationship 11%
• Protein Kinase 11%
• Double mutant 11%
• Cellular Response 11%
• Ataxia Telangiectasia 11%
• Immunodeficiency 11%
• DNA Damage 11%
• Genomic Instability 11%
• DNA Double-strand Breaks 11%
• Ionizing Radiation 11%
• ATM Protein 11%
• Double-strand Breaks 11%
• Mouse Strain 11%
• Crosslinking Agent 11%
• Short Lifetime 11%
• DNA Damage Response 11%
• Progressive Ataxia 11%
• Cancer Predisposition 11%
• Reduced Body 11%
• Non-homologous End Joining 11%
• Neurological Phenotype 11%
• Mutant Genotypes 11%
• Mitomycin C 11%
• Compound Genotype 11%
• Differential Interactions 11%
• Combined Inactivation 11%
• IR Sensitivity 11%
• Mendelian Ratio 11%
• Radiation Sensitivity 11%

Biochemistry, Genetics and Molecular Biology

• Paralogy 100%
• Homologous Recombination 100%
• Recombination Repair 100%
• Erethism 40%
• Mouse 20%
• Cross-Link 20%
• Protein Kinases 20%
• Lifespan 20%
• Body Weight 20%
• DNA Damage Response 20%
• Genome Instability 20%
• Radiosensitivity 20%
• Mouse Strain 20%
• Non-Homologous End Joining 20%