Acetylcholine receptor gene expression in experimental autoimmune myasthenia gravis

Orna Asher, Drorit Neumann, Veit Witzemann, Sara Fuchs*

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

21 Scopus citations

Abstract

Acetylcholine receptor (AChR) gene expression was analyzed in experimental autoimmune myasthenia gravis (EAMG) in rabbits, rats and mice. An increase in AChR transcripts was demonstrated to be exclusively associated with myasthénie symptoms and with a severe loss in membrane AChR. An increase of α-, β-, ε{lunate}-, and δ-subunit specific mRNAs (5.2-, 1.6-, 3.2- and 3.7-fold, respectively), which code for the adult type of AChR (α2βε{lunate}δ) was observed in EAMG in rats. The γ-subunit transcript was not detectable in myasthenie or healthy rats. It appears that the regulatory control of AChR gene expression in EAMG is different from that observed upon denervation.

Original languageEnglish
Pages (from-to)231-235
Number of pages5
JournalFEBS Letters
Volume267
Issue number2
DOIs
StatePublished - 16 Jul 1990
Externally publishedYes

Funding

FundersFunder number
AssociationF rancaiseC ontrel es Myopathies
Muscular DystrophyA ssociationof America
MyastheniaG ravis Foundation
United States-IsraeBl inationalS cienceF oundation
Association Française contre les Myopathies
United States-Israel Binational Science Foundation

    Keywords

    • Adult and embryonic acetylcholine receptor
    • Experimental autoimmune myasthenia gravis
    • Subunit transcript

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