A longitudinal examination of the neurodevelopmental impact of prenatal immune activation in mice reveals primary defects in dopaminergic development relevant to schizophrenia

Keyphrases

• Adult Stage 50%
• Attenuation 50%
• Developmental Neuropathology 50%
• Dopamine 50%
• Dopamine Dysfunction 50%
• Dopamine Function 50%
• Dopamine System 50%
• During Pregnancy 50%
• Early Intervention 50%
• Environmental Risk Factors 100%
• Fetal Stage 50%
• Functional Abnormalities 50%
• Immune Challenge 50%
• Intervention Targets 50%
• Life Stages 100%
• Longitudinal Assessment 100%
• Longitudinal Investigation 50%
• Maldevelopment 50%
• Mouse Model 50%
• Neurodevelopmental 100%
• Neuropathology 50%
• Polyinosinic-polycytidylic Acid 50%
• Prenatal Exposure 100%
• Prenatal Immune Activation 100%
• Prenatal Immune Challenge 100%
• Primary Defects 100%
• Psychosis 100%
• Psychotic Disorders 50%
• Related Disorders 50%
• Schizophrenia 100%
• Schizophrenia Symptoms 50%
• Viral 50%

Neuroscience

• Behavior (Neuroscience) 14%
• Dementia Praecox 100%
• Dopaminergic 100%
• Ontogeny 14%
• Polyinosinic:polycytidylic Acid 14%
• Psychosis 14%